Nothing magical about it; just simple chemistry. I gave you a ton of references (one from pubmed and more than a dozen in the long article I posted) that lend credence to the theory. Test after test has shown that nandrolone has a weaker binding constant for aromatase than test by at least an order of magnitude. Any test present will preferentially bind. Yet, deca aromatizes at half the rate of test. How is that possible if the binding constant for aromatase is more than 10 times less? There has to be another mechanism at play.
It is actually your theory (and that of many others) that progesterone and/or prolactin can cause gyno even in the absence of estrogen that lacks any proof. Find me some articles that support your theory. I found some that support mine.
I was saying that a-dex wouldn't touch the estro effects from deca. Yes of course it will stop the aromatization of the test but you are going to have much more deca present than test, right?
If you insist on being a guinea pig, have an open mind. Your previous experience with tren gyno supports my theory as well. More a-dex didn't help. Would caber have helped? I don't know.
I guess my point is: If you really want to provide support for your theory that progesterone or prolactin causes gyno, you would have that opportunity if you started to experience gyno symptoms. If caber, a progesterone/prolactin inhibitor, relieves the symptoms, that is evidence you are right.
It is actually your theory (and that of many others) that progesterone and/or prolactin can cause gyno even in the absence of estrogen that lacks any proof. Find me some articles that support your theory. I found some that support mine.
I was saying that a-dex wouldn't touch the estro effects from deca. Yes of course it will stop the aromatization of the test but you are going to have much more deca present than test, right?
If you insist on being a guinea pig, have an open mind. Your previous experience with tren gyno supports my theory as well. More a-dex didn't help. Would caber have helped? I don't know.
I guess my point is: If you really want to provide support for your theory that progesterone or prolactin causes gyno, you would have that opportunity if you started to experience gyno symptoms. If caber, a progesterone/prolactin inhibitor, relieves the symptoms, that is evidence you are right.

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